AP-1

GPTKB entity

Statements (64)
Predicate Object
gptkbp:instance_of gptkb:DNA
gptkbp:associated_with gptkb:Oncology
gptkbp:composed_of gptkb:Fos_protein
gptkb:Jun_protein
gptkbp:has_role immune response
https://www.w3.org/2000/01/rdf-schema#label AP-1
gptkbp:influences inflammation
gptkbp:initiated_by growth factors
gptkbp:interacts_with gptkb:c-Fos
gptkb:c-Jun
gptkbp:involved_in gptkb:diabetes
gptkb:developers
gptkb:neurodegenerative_diseases
toxicology
aging
apoptosis
cell cycle regulation
cell proliferation
cellular homeostasis
cellular stress response
chromatin remodeling
circadian rhythms
signal transduction
autoimmune diseases
metabolic syndrome
obesity
wound healing
neuroplasticity
stress response
fibrosis
stem cell biology
cardiovascular diseases
cellular senescence
neuronal signaling
oxidative stress response
synaptic plasticity
tumor microenvironment
cellular differentiation
differentiation
atherosclerosis
tissue repair
viral infections
cell migration
metastasis
bacterial infections
transcriptional regulation
tumor progression
epigenetic regulation
drug metabolism
learning and memory
therapeutic resistance
reproductive biology
pain signaling
hormonal signaling
hypoxia response
chronic inflammatory diseases
allergic responses
environmental stress response
gptkbp:is_found_in gptkb:Atom
gptkbp:is_regulated_by gptkb:mitogen-activated_protein_kinase_(MAPK)
gptkbp:regulates gene expression
gptkbp:bfsParent gptkb:A-8_Motorway
gptkb:A-12_Motorway
gptkbp:bfsLayer 6