Statements (69)
| Predicate | Object |
|---|---|
| gptkbp:instanceOf |
protein
|
| gptkbp:associatedWith |
cancer progression
|
| gptkbp:diedIn |
vascular permeability
|
| gptkbp:established |
VEGF binding
|
| gptkbp:function |
regulates angiogenesis
|
| gptkbp:hasPerformedAt |
gptkb:VEGFR-1
VEGFR-2 VEGFR-3 |
| https://www.w3.org/2000/01/rdf-schema#label |
VEGFR
|
| gptkbp:involvedIn |
tumor growth
wound healing |
| gptkbp:is_evaluated_by |
endothelial cells
|
| gptkbp:isActiveIn |
VEGFR_inhibitors
|
| gptkbp:isAssociatedWith |
diabetic retinopathy
|
| gptkbp:isInvolvedIn |
apoptosis regulation
cell differentiation cell proliferation cellular stress response immune response protein synthesis signal transduction muscle development embryonic development bone healing cell survival cellular aging extracellular matrix remodeling organ development inflammatory responses neural development atherosclerosis gene expression regulation ischemia placental development tissue repair neuroprotection cell migration metastasis stem cell maintenance testis development retinal diseases tissue homeostasis cell fate determination hair follicle development metabolic regulation adipose tissue development heart development lung development pancreas development placenta development neovascularization ovary development germ cell development skin development cell-cell communication gastrointestinal development liver development lymphangiogenesis spleen development thymus development kidney_development |
| gptkbp:isLinkedTo |
cardiovascular diseases
|
| gptkbp:isLocatedIn |
various tissues
|
| gptkbp:isRegulatedBy |
hypoxia
|
| gptkbp:isStudiedIn |
clinical trials
|
| gptkbp:isTrainedIn |
anti-angiogenic therapy
|
| gptkbp:location |
cell membrane
|
| gptkbp:related_to |
vascular endothelial growth factor (VEGF)
|
| gptkbp:type |
receptor tyrosine kinase
|