Statements (71)
| Predicate | Object |
|---|---|
| gptkbp:instance_of |
gptkb:vaccine
|
| gptkbp:bfsLayer |
5
|
| gptkbp:bfsParent |
gptkb:ALX-0701
|
| gptkbp:associated_with |
immune response
|
| gptkbp:discovered_by |
gptkb:Robert_G._Miller
|
| gptkbp:enhances |
cytokine production
|
| gptkbp:established |
apoptosis of T cells
|
| gptkbp:first_introduced |
gptkb:1980s
|
| gptkbp:function |
stimulates T cell proliferation
enhances B cell growth promotes the growth of natural killer cells |
| gptkbp:habitat |
chromosome 4
|
| https://www.w3.org/2000/01/rdf-schema#label |
IL-2
|
| gptkbp:influences |
gptkb:psychologist
fertility mental health reproductive health cellular metabolism circadian rhythms energy homeostasis glucose metabolism lipid metabolism child development chronic diseases cognitive function wound healing neuroinflammation fungal infections oxidative stress response sleep regulation tumor microenvironment cellular aging immune tolerance chronic inflammation immune cell activation aging process tissue repair viral infections hormonal regulation stress responses autoimmunity bacterial infections graft rejection behavioral responses protein metabolism metabolic regulation parasitic infections transplantation outcomes lactation vaccine responses effector T cells memory T cells immune checkpoint regulation pregnancy outcomes allergic responses stem cell function cytokine signaling pathways T regulatory cells |
| gptkbp:is_involved_in |
inflammation
humoral immunity T cell differentiation cell-mediated immunity immune regulation |
| gptkbp:is_vulnerable_to |
gptkb:IL-2_receptor
|
| gptkbp:produced_by |
gptkb:T_cells
lymph nodes spleen |
| gptkbp:research_areas |
gptkb:healthcare_organization
autoimmune diseases treatment |
| gptkbp:security_features |
activated T cells
|
| gptkbp:used_in |
gptkb:vaccine
|