Properties (69)
| Predicate | Object |
|---|---|
| gptkbp:instanceOf |
nucleoside
|
| gptkbp:aimsTo |
cognitive function
neuroprotection |
| gptkbp:associated_with |
cancer progression
|
| gptkbp:can_be |
adenosine receptors
a neurotransmitter |
| gptkbp:communitySupport |
pain perception
vascular tone glutamate release |
| gptkbp:composedOf |
adenine
ribose |
| gptkbp:enables |
neurotransmitter release
|
| gptkbp:has_a_role_in |
energy transfer
sleep regulation |
| https://www.w3.org/2000/01/rdf-schema#label |
adenosine
|
| gptkbp:impact |
heart rate
immune cell function mood and behavior vascular_health |
| gptkbp:influence |
cellular differentiation
vascular permeability sleep-wake cycles |
| gptkbp:is_a |
the immune system
the central nervous system |
| gptkbp:is_a_key_component_of |
gptkb:ADP
gptkb:ATP AMP NADH cardiovascular health |
| gptkbp:is_a_key_player_in |
metabolic adaptation
cellular energy homeostasis metabolic stress response |
| gptkbp:is_a_platform_for |
adenosine deaminase
|
| gptkbp:is_a_promoter_of |
immune response
neurotransmitter systems |
| gptkbp:is_available_in |
all living cells
|
| gptkbp:is_involved_in |
cell proliferation
metabolic disorders neurodegenerative diseases signal transduction neuroinflammation cell signaling pathways synaptic plasticity angiogenesis neurotransmission vasodilation metabolic regulation pain modulation cellular signaling networks cellular apoptosis neuroprotection mechanisms neurotransmitter recycling hypoxia-induced signaling inflammation processes |
| gptkbp:is_used_in |
treatment of arrhythmias
cardiac stress tests |
| gptkbp:measures |
blood plasma
|
| gptkbp:produces |
hypoxia
ATP_breakdown dephosphorylation_of_ATP |
| gptkbp:regulates |
gene expression
inflammatory responses |
| gptkbp:targets |
pharmacological agents
anti-inflammatory drugs cancer therapies |
| gptkbp:transferFee |
inosine
|
| gptkbp:was_a_catalyst_for |
cAMP
|
| gptkbp:was_a_factor_in |
tissue repair
cardioprotection |