Statements (73)
| Predicate | Object |
|---|---|
| gptkbp:instance_of |
gptkb:physicist
|
| gptkbp:bfsLayer |
4
|
| gptkbp:bfsParent |
gptkb:ANSI_Common_Lisp
|
| gptkbp:associated_with |
gptkb:historical_event
schizophrenia anxiety disorders excitatory signaling |
| gptkbp:community |
calcium ions
|
| gptkbp:established |
gptkb:superhero
|
| gptkbp:function |
neurotransmission
|
| gptkbp:has_role |
gptkb:Magic:_The_Gathering_set
gptkb:learning |
| https://www.w3.org/2000/01/rdf-schema#label |
CL-GLUT
|
| gptkbp:implements |
agonists
|
| gptkbp:interacts_with |
glutamate
|
| gptkbp:is_involved_in |
cell signaling
cellular homeostasis cellular stress response circadian rhythms immune response metabolic processes neurotransmitter release signal transduction metabolic syndrome neuroinflammation stress response cardiovascular health cognitive functions cellular proliferation cell survival pain perception sleep regulation synaptic plasticity cell death cellular differentiation motor control oxidative stress aging process neuroprotection cell adhesion hormonal regulation cellular signaling pathways appetite regulation development of the nervous system synaptic transmission neural plasticity neurite outgrowth neurotransmitter synthesis behavioral responses long-term potentiation long-term depression cellular repair mechanisms glial cell function axon guidance dendritic growth cellular apoptosis neurotransmitter recycling neurotransmitter transport neurotransmitter degradation receptor internalization neural circuit formation cell-cell communication neurotrophic factor signaling neurotransmitter uptake receptor trafficking synaptic vesicle cycle ion channel regulation synaptic remodeling extracellular matrix interactions receptor desensitization |
| gptkbp:is_represented_in |
gptkb:microprocessor
|
| gptkbp:located_in |
central nervous system
|
| gptkbp:related_to |
gptkb:psychologist
|